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Amlop (Atenolol)

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Amlop is used with or without other medications to treat high blood pressure (hypertension). Lowering high blood pressure helps prevent strokes, heart attacks, and kidney problems.

Other names for this medication:
Agloten, Alinor, Amloat, Amlobid, Amlogla, Amloman, Amlonol, Amlop, Amloray, Amlosac, Amlosafe, Amlotec, Amloz, Amsten, Anten, Atecard, Atekind, Atemide, Atenex, Cardica, Cardipin, Cardipress, Celbeta, Lodol, Lonol, Nipidol, Odinol, Ortibid, Ozoten, Systonorm, Tecard, Tenase, Tenil, Tenlol, Tensicard, Tentoric

Similar Products:
Neurontin, Actos, Duricef, Cytoxan, Compazine, Amaryl, Zovirax, Assepium, Nolcer, Tibitol, Femidin, Actamide,

Also known as: Atenolol


Amlop (atenolol) is a beta-blocker that affects the heart and circulation (blood flow through arteries and veins).

Amlop is used to treat angina (chest pain) and hypertension (high blood pressure).

Amlop is also used to lower the risk of death after a heart attack.


A nurse or other trained health professional will give you this medicine. This medicine is given through a needle placed in one of your veins .

Detailed Amlop dosage information


Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Overdose symptoms may include extreme weakness or lack of energy, very slow heart rate, shortness of breath, or fainting.


Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Keep container tightly closed. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Amlop are:

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A new technique for anastomosing veins to small-caliber arteries. Clinical, enzymatic, and pathological study of an infant with the acute form of the disease. The radial artery forearm flap: an anomaly of the radial artery. Lipoxygenase (LOX)-catalyzed cooxidation of the human carcinogen benzidine (BZD) has been shown in in vitro enzyme systems. The pharmacological effects of some radioprotective agents in mice. The changes can be carried out reversibly without injury to the cell. In this study we determined the effects of proteasome inhibition on surfactant protein expression in H441 and MLE-12 lung epithelial cells to understand the relationship between proteasome dysfunction and surfactant protein gene expression. The addition of sodium dodecyl sulfate (SDS) to the keratin solution prior to dialysis prevented extensive aggregation of the keratin chains.

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